<mets:mets OBJID="eprint_11141" LABEL="Eprints Item" xsi:schemaLocation="http://www.loc.gov/METS/ http://www.loc.gov/standards/mets/mets.xsd http://www.loc.gov/mods/v3 http://www.loc.gov/standards/mods/v3/mods-3-3.xsd" xmlns:mets="http://www.loc.gov/METS/" xmlns:mods="http://www.loc.gov/mods/v3" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance"><mets:metsHdr CREATEDATE="2023-07-05T18:24:24Z"><mets:agent ROLE="CUSTODIAN" TYPE="ORGANIZATION"><mets:name>OAR@ICRISAT</mets:name></mets:agent></mets:metsHdr><mets:dmdSec ID="DMD_eprint_11141_mods"><mets:mdWrap MDTYPE="MODS"><mets:xmlData><mods:titleInfo><mods:title>Insights Into the Nitric Oxide Mediated Stress Tolerance in Plants</mods:title></mods:titleInfo><mods:name type="personal"><mods:namePart type="given">P</mods:namePart><mods:namePart type="family">Santisree</mods:namePart><mods:role><mods:roleTerm type="text">author</mods:roleTerm></mods:role></mods:name><mods:name type="personal"><mods:namePart type="given">S S</mods:namePart><mods:namePart type="family">Adimulam</mods:namePart><mods:role><mods:roleTerm type="text">author</mods:roleTerm></mods:role></mods:name><mods:name type="personal"><mods:namePart type="given">K</mods:namePart><mods:namePart type="family">Sharma</mods:namePart><mods:role><mods:roleTerm type="text">author</mods:roleTerm></mods:role></mods:name><mods:name type="personal"><mods:namePart type="given">P</mods:namePart><mods:namePart type="family">Bhatnagar-Mathur</mods:namePart><mods:role><mods:roleTerm type="text">author</mods:roleTerm></mods:role></mods:name><mods:name type="personal"><mods:namePart type="given">K K</mods:namePart><mods:namePart type="family">Sharma</mods:namePart><mods:role><mods:roleTerm type="text">author</mods:roleTerm></mods:role></mods:name><mods:abstract>During the last two decades, several studies have established nitric oxide (NO) as a crucial signaling molecule during plant stress responses. NO protect plants from stressful conditions mostly through the activation of antioxidant defense, by maintaining metabolic homeostasis, by altering the gene transcription and posttranslational protein modifications. So far, most of the NO functions have been explored based on manipulation of endogenous NO levels by exogenous donors/scavengers or through mutants and transgenics. However, it is hard to draw any clear conclusions, since most of these studies are not uniform, being rather superficial without exploring the underlying signaling pathways. Indeed, the integration of the crosstalk events between NO and other signaling molecules under stress responses is also very critical. Importantly, lack of complete understanding of its production and signaling cascade is a serious setback for further elucidation by genetic and molecular approaches. Therefore, a step forward now will be to explore more NO responsive genes, proteins, and their networks under stress to serve as a key resource for further NO research.</mods:abstract><mods:classification authority="lcc">Abiotic Stress</mods:classification><mods:classification authority="lcc">Plant Genetics</mods:classification><mods:originInfo><mods:dateIssued encoding="iso8061">2019</mods:dateIssued></mods:originInfo><mods:originInfo><mods:publisher>Elsevier</mods:publisher></mods:originInfo><mods:genre>Book Section</mods:genre></mets:xmlData></mets:mdWrap></mets:dmdSec><mets:amdSec ID="TMD_eprint_11141"><mets:rightsMD ID="rights_eprint_11141_mods"><mets:mdWrap MDTYPE="MODS"><mets:xmlData><mods:useAndReproduction>
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